Exogenous itaconate addition in apolipoprotein E-deficient (Apoe−/−) mice suppressed the initiation and progression of AAA and downstream inflammatory protein although Irg1 deficiency reverted the influence of inhibitory. Overexpression of Keap1 or transferred Cys151S mutant Keap1 vector also abolished the activation of Nrf2 induced by itaconat
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Effectiveness with a discrimination task requiring matching of colors introduced sequentially with a computer screenA weakness of our methodology was that the next reviewer was restricted to independently screening 20% of titles, abstracts and whole textual content papers and extracting info from 20% of provided studies resulting from constrained m